Prevalence of venous obstructions in (recurrent) venous thromboembolism: a case-control study

Thrombosis Journal

Table 2 Details in patients with central venous obstructions and anatomic anomalies

Recurrent VTE N = 32		No recurrent VTE N = 24		Total N = 56
6 (18.8)		5 (20.8)		11 (19.6)
Anatomic anomalies
#1	Duplication of the VP, fibrosis of the VF	#1	Aneurysm VP
#2	Duplication of the VF	#2	Duplication and fibrosis of the VF
Central venous obstructions
#3	Extraluminal compression: CIV and EIV	#3	Extraluminal compression: ICVir and CIV
#4	Extraluminal compression: CIV^a	#4	Extraluminal compression: ICVir and CIV
#5	Extraluminal compression: ICVir and CIV^b	#5	Extraluminal compression: CIV^c
#6	Extraluminal compression: CIV^c

Data are n (%)
ICVir Inferior caval vein, infra renal, CIV Common iliac vein, EIV External iliac vein, FV Femoral vein, PV Popliteal vein, VTE Venous thrombo-embolism
None of the variables mentioned in this table showed statistical significant difference between groups
Venous obstruction is defined as either extraluminal compression (e.g. due to May-Thurner Syndrome, adjacent anatomical structures, pelvic tumour) or the presence of anatomical anomalies (e.g. agenesis, hypoplasia, aneurysms, anatomical variances, and duplications) that might negatively influence the central venous flow
^a Extraluminal compression caused by spondylosis
^b Extraluminal compression caused by the left iliac artery
^c Extraluminal compression caused by May Thurner Syndrome (compression by the right iliac artery)

ISSN: 1477-9560